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NYU Langone Provider

Edward A. Fisher, MD, MPH, PhD

NYU Langone Provider
  • Specialties: Preventive Cardiology, Cardiology
  • Treats: Adults
  • Language: English
  • Phone: 212-263-0855
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Board Certifications
  • American Board of Pediatrics - Pediatrics, 1981
Education and Training
  • Fellowship, NIH - National Institute of Health, Medical Genetics, 1984
  • Fellowship, Boston Children's Hospital, Gastroenterology, 1982
  • PhD from Massachusetts Inst of Tech, 1982
  • MPH from UNC-Chapel Hill, 1978
  • Residency, Duke University Hospital, Pediatric Medicine, 1977
  • MD from New York University, 1975

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This provider accepts the following insurance plans.

  • Aetna
     
    • Aetna HMO
    • Aetna Indemnity
    • Aetna Medicare
    • Aetna POS
    • Aetna PPO/EPO
  • Blue Cross Blue Shield
     
    • Empire BCBS
    • Empire BCBS Top Tier
  • Cigna
     
    • Cigna EPO/POS
    • Cigna PPO
  • Emblem
     
    • Emblem Select Care Exchange
  • Empire Blue Cross Blue Shield
     
    • Empire Blue Cross Blue Shield EPO
    • Empire Blue Cross Blue Shield HMO
    • Empire Blue Cross Blue Shield HealthPlus
    • Empire Blue Cross Blue Shield Indemnity
    • Empire Blue Cross Blue Shield MediBlue
    • Empire Blue Cross Blue Shield POS
    • Empire Blue Cross Blue Shield PPO
  • GHI
     
    • GHI CBP
    • GHI HMO
  • HIP
     
    • HIP Access I
    • HIP Access II
    • HIP Child Health
    • HIP EPO/PPO
    • HIP HMO
    • HIP Medicaid
    • HIP Medicare
    • HIP POS
View All Accepted Plans This list of insurances changes regularly, and insurance plans listed may not be accepted at all office locations for this provider. Before your appointment, please confirm with your insurance company that this provider accepts your insurance.

Edward A. Fisher, MD, MPH, PhD does not accept insurance.

Locations and Appointments

NYU Langone Center for the Prevention of Cardiovascular Disease

530 First Avenue, Suite 4F, New York, NY 10016

Phone

212-263-0855

Fax

646-501-0150

Interests

Atherosclerosis, Macrophages, Inflammation, Lipoproteins, Diabetes, Obesity

Research Summary

Molecular and immunologic determinants of the factors causing atherosclerosis and cardiovascular disease

Our laboratory has a longstanding interest in two major areas. One is the cell biology of the assembly and secretion of the macromolecular complexes that transport lipids (mainly triglycerides and cholesterol) made in the liver to other tissues, where they are used for a number of essential purposes. Some of the strongest risk factors for cardiovascular disease—the leading killer not just in the U.S., but worldwide—are related to the levels of certain lipoproteins in the blood. Thus, information about the mechanisms on how they are formed is of great value in understanding how to regulate the levels of the cardiovascular disease-causing lipoproteins. Recently, we have discovered factors that coregulate lipoprotein assembly and secretion and the formation of the types of lipid droplets that can accumulate to cause “fatty liver”, a serious and increasingly common health problem associated with obesity.

The other major research area is the underlying cause of cardiovascular disease, namely atherosclerosis, the buildup of cholesterol-filled cells that form plaques, such as in the coronary arteries. These plaques can rupture and cause a heart attack. We have pioneered mouse models of atherosclerosis and molecular approaches so that we can discover the key factors that will return a diseased artery back to a healthier state. We have discovered that reducing the levels in the blood of certain lipoproteins coupled with resolving the inflammation of the immune cells in the plaques is the optimal combination to achieve this. We are extending these studies to the molecular level to identify specific therapeutic targets as well as to human plaques and to clinically relevant conditions known to increase plaque inflammation and the risk of cardiovascular disease, such as obesity, insulin resistance, and diabetes.

These focus areas and their associated publications are derived from PubMed and the MeSH term library. *
represents one publication
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*Due to PubMed processing times, the most recent publications may not be reflected in the timeline.

Read All Publications (430)

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