Ira Goldberg at [NYU Langone Medical Center]

Ira J. Goldberg, MD

  • Specialty: Endocrinology
  • Language: English
  • Phone: 212-481-1350

About Me

Conditions and Treatments

lipid disorders, familial hypercholesterolemia, high cholesterol level, metabolic syndrome, hypocholesterolemia, type 2 diabetes mellitus, hypothyroidism, thyroid diseases, graves' disease, hyperthyroidism, pregnancy induced diabetes, calcium disorders, prediabetes, chronic thyroiditis, combined hyperlipidemia, hypoparathyroidism, high triglycerides, diabetes mellitus complications, thyroiditis, polygenic hypercholesterolemia, multiple endocrine neoplasia, hyperparathyroidism, insulin dependent diabetes, postpartum thyroiditis, osteoporosis, parathyroid disease, nutritional therapy, silent thyroiditis, metabolic bone disease, autoimmune thyroid disease
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Credentials

Positions
  • Clarissa and Edgar Bronfman, Jr. Professor of Endocrinology, Department of Medicine
  • Director, Division of Endocrinology
Board Certifications
  • American Board of Internal Medicine (Endocrinology, Diabetes & Metabolism), 1981
  • American Board of Internal Medicine - Internal Medicine, 1978
Education and Training
  • MD from Harvard Medical School, 2014
  • Fellowship, Mount Sinai Medical Center (New York, NY), Endoc & Metabolism, 1979
  • Residency, Bellevue Hospital Center, Medicine, 1978
Departments

Locations and Appointments

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NYU Diabetes & Endocrine Associates

530 1st Avenue, Suite 5E
New York, NY 10016

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My Research

Dr. Goldberg's research has focused on abnormalities of lipoprotein metabolism, macrovascular disease in diabetes, and the role of triglycerides in atherosclerosis. He has received grant support in a number of investigational studies that involve atherogenicity of apolipoprotein B-containing lipoproteins, regulation of plasma triglyceride by lipase enzymes, diabetic macrovascular disease, and lipid uptake and toxicity in the heart. Initial work in Dr. Goldberg's laboratory focused on the cellular biology and physiological function of lipoprotein lipase (LpL). He is widely acknowledged to be one to the worlds experts in lipid uptake pathways needed to provide fuel for the heart. These studies funded by the NIH have shown the central role of triglycerides in heart energy metabolism and, in turn, the role of the heart in regulating circulating levels of triglyceride-rich lipoproteins.

Excess lipid accumulation within tissues leads to dysfunction, a condition often referred to as lipotoxicity. Lipotoxicity is widely believed to create insulin-deficient diabetes, non-alcoholic fatty liver disease (NAFLD), and some forms of heart failure. The Goldberg laboratory has studied lipotoxicity in the heart and created two novel models, one with overexpression of cardiomyocyte-anchored LpL and a second with overexpression of PPARg. The Goldberg laboratory has been the leading laboratory in developing methods to rescue cardiac lipotoxicity and has studied methods to reduce cardiac ceramide production, increase conversion of toxic lipids to triglyceride using overexpression of diacylglycerol acyl transferase 1 (DGAT1), and altering lipid uptake using LpL deletion, and reducing ceramide production.

In addition to this research, the laboratory has also studied mechanisms of atherogenesis in diabetic mice. Part of this activity is funded by a Dual-PI grant along with Dr. Edward Fisher. Together they have shown that hyperglycemia leads to defective regression of atherosclerosis, i.e. resolution of vascular disease after the marked reduction of circulating cholesterol. These studies are meant to mimic the remodeling of the artery in patients with statin treatment. Using sodium glucose co-transporter 2 (SGLT2) inhibition, the Goldberg and Fisher laboratories have shown that hyperglycemia and obesity lead to inflammatory white blood cells and bone marrow stimulation. Additionally, Dr. Goldberg is a Project Leader and Dr. Fisher a Co-Investigator on The Cardiovascular Complications and Diabetes Program Project at the University of Washington directed by Dr. Karin E. Bornfeldt (http://depts.washington.edu/doce/the-pathobiology-of-macrovascular-disease-in-diabetes-program-project/).

Publications

  • CD36 actions in the heart: Lipids, calcium, inflammation, repair and more?

    Abumrad, Nada A; Goldberg, Ira J Abumrad, Nada A; Goldberg, Ira J
    Biochimica & biophysica acta. 2016 Mar 21. 1860 (10): 1442-1449

  • Triglyceride Treatment in the Age of Cholesterol Reduction

    Agrawal, Nidhi; Corradi, Patricia Freitas; Gumaste, Namrata; Goldberg, Ira J Agrawal, Nidhi; Corradi, Patricia Freitas; Gumaste, Namrata; Goldberg, Ira J
    Progress in cardiovascular diseases. 2016 Sep -. 59 (2): 107-118

  • LRP-1 deficiency in type II pneumocytes decreased surfactant but increased intracellular lipids [Meeting Abstract]

    Garcia-Arcos, I; Goldberg, I J; Foronjy, R F Garcia-Arcos, I; Goldberg, I J; Foronjy, R F
    FASEB journal. 2016 April 2016. 30 (no):

Read All Publications (196)