When I came to NYU Langone as a resident in the 1970s, it was—and still is—an outstanding medical school and hospital with wonderful people. The longer I stayed, the more I wanted to become part of its close-knit, interdisciplinary community of physicians and scientists. I still carry on the tradition that goes back to 1841, when NYU School of Medicine was founded: I’m dedicated to treating a diverse group of people, and to putting patients first.
I chose internal medicine because of my interest in a broad range of diseases. Rheumatology is my specialty, because I am especially concerned with how little we understand about immunological conditions, in which the body attacks itself.
Whether researching new treatments for heart disease or rheumatoid arthritis, I’m not only focused on the science but on making medical advances that have direct implications for my patients’ health.
- Frederick H. King Professor of Internal Medicine, Department of Medicine
- Professor, Department of Pathology
- Chair of the Department of Medicine
- Vice Dean for Education Faculty & Academic Affair
- American Board of Internal Medicine (Rheumatology), 1980
- American Board of Internal Medicine - Internal Medicine, 1977
Education and Training
- Fellowship, Bellevue Hospital, Rheumatology, 1986
- Fellowship, Bellevue Hospital, Rheumatology, 1983
- Fellowship, Bellevue Hospital, Medicine - Rheumatology, 1980
- MD from Harvard Medical School, 1974
Locations and Appointments
- HealthSmart (WTC)
- UnitedHealthcare Medicare
- UnitedHealthcare Top Tier
“We’re investigating the genetic causes of many conditions, and our findings are improving patients’ lives.”Steven Abramson, MD Rheumatologist
Furthermore, our laboratory has long standing interest in the functions for nitric oxide (NO) and prostaglandin E2 in the activation of MAP kinase signaling, protease synthesis and cellular death in chondrocyte and synovial fibroblast. Recently, we identified a novel latent tumor growth factor-beta1 (TGF-beta1) activating an extracellular matrix protein F-spondin. F-spondin has not been studied in differentiation of chondrocyte, endochondral ossification, cartilage matrix metabolism and pathological mineralization process. We are planning to develop an F-spondin knock out mouse to explore the functions of F-spondin and TGFb-1 in the development of OA. The fundamental knowledge obtained from these studies is crucial for understanding the pathogenesis and treatment of diseases such as osteoarthritis and rheumatoid arthritis.
Relationship between meniscal integrity and risk factors for cartilage degeneration
Arno, Sally; Bell, Christopher P; Xia, Ding; Regatte, Ravinder R; Krasnokutsky, Svetlana; Samuels, Jonathan; Oh, Cheongeun; Abramson, Steven; Walker, Peter S
Knee. 2016 May 11. ?-?
ROLE OF PERIOSTIN AND DISCOIDIN DOMAIN RECEPTOR-1 (DDR1) IN THE REGULATION OF CARTILAGE DEGENERATION AND EXPRESSION OF MMP-13 [Meeting Abstract]
Attur, M; Yang, Q; Kirsch, T; Abramson, SB
Osteoarthritis & cartilage. 2016 APR. 24: S156-S156
When is osteonecrosis not osteonecrosis? adjudication of reported serious adverse joint events in the tanezumab clinical development program
Hochberg, Marc C; Tive, Leslie A; Abramson, Steven B; Vignon, Eric; Verburg, Kenneth M; West, Christine R; Smith, Mike D; Hungerford, David S
Arthritis & rheumatology. 2016 Feb . 68 (2): 382-391